现在因癌症死亡的人数终于超过了心血管疾病。经常看到周遭的朋友患癌而去，新闻上看到名人富人白人黑人都不能幸免。大家真是谈癌色变。我的母亲也是因癌症而去世的。想到得癌症后的开刀，放疗，化疗所带来生不如死的痛苦，其恐惧感不可想象。
随着科学家们对于癌症的不断研究，最近终于从分子生物学的角度对于癌症的生长过程有了突破性的发现。美国哈佛大学医学院发现了控制癌细胞生长的分子开关。如果对于该分子开关进行控制，那么癌细胞可以转变成正常细胞。
尽 管当今癌症理论学派无数，但归根到底癌症是个代谢病。糖尿病是个代谢病，而癌症实际是个另类糖尿病。糖尿病与癌症同属代谢病，是两个不同的极端。从细胞层 次上讲，糖尿病是细胞失去了使用糖的能力，由于缺乏能量而慢慢退化，最后导致器官衰竭。癌症是细胞无 节制地吸入糖导致细胞无节制生长，最后导致破坏器官的正常功能。对于糖尿病病人得癌症后死亡率很高。好消息是癌症和糖尿病的治疗方法基本相同，因为同为代 谢病，其平衡点是一样的。
抗癌总愿则：少吃米面，多吃素菜，适量荤食。达到和保持弱碱体质。保持血液干净，营养充足。跟据最新研究发现，四十岁以上者体内都有微型癌肿瘤，在大量补充抗氧剂的情况下，微型癌肿瘤不会长大，最后会消失。在肿瘤消失时，其大小便会有强烈的臭味。
我将这几年帮助他人调理的体会整理了一下同大家分享和讨论。
中文讲座的连接为，http://www.tudou.com/programs/view/0OVMVM793gI/
英文讲座的连接为，http://www.tudou.com/programs/view/p2vVRAtRMWQ/
声明，我不是医生，这个讲座是教育性质的。有病看医生噢。
参考资料，
http://www.sciencemag.org/cgi/content/abstract/324/5930/1029
Understanding the Warburg Effect: The Metabolic Requirements of Cell Proliferation
Matthew G. Vander Heiden,1,2 Lewis C. Cantley,2,* Craig B. Thompson3,*
In contrast to normal differentiated cells, which rely primarily on mitochondrial oxidative phosphorylation to generate the energy needed for cellular processes, most cancer cells instead rely on aerobic glycolysis, a phenomenon termed "the Warburg effect." Aerobic glycolysis is an inefficient way to generate adenosine 5'-triphosphate （ATP）, however, and the advantage it confers to cancer cells has been unclear. Here we propose that the metabolism of cancer cells, and indeed all proliferating cells, is adapted to facilitate the uptake and incorporation of nutrients into the biomass （e.g., nucleotides, amino acids, and lipids） needed to produce a new cell. Supporting this idea are recent studies showing that （i） several signaling pathways implicated in cell proliferation also regulate metabolic pathways that incorporate nutrients into biomass; and that （ii） certain cancer-associated mutations enable cancer cells to acquire and metabolize nutrients in a manner conducive to proliferation rather than efficient ATP production. A better understanding of the mechanistic links between cellular metabolism and growth control may ultimately lead to better treatments for human cancer.
1 Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA 02115, USA.
2 Beth-Israel Deaconess Cancer Center and Department of Systems Biology, Harvard Medical School, Boston, MA 02115, USA.
3 Department of Cancer Biology, Abramson Cancer Center, University of Pennsylvania, Philadelphia, PA 19104 USA.
http://www.sciencedaily.com/releases/2010/01/100122222228.htm
Potential Way to Reverse Cancer Cell Metabolism and Tumor Growth
ScienceDaily （Jan. 25, 2010） — A team of scientists led by Professor Adrian Krainer, Ph.D., of Cold Spring Harbor Laboratory has discovered molecular factors in cancer cells that boost the production of an enzyme that helps alter the cells' glucose metabolism. The altered metabolic state, called the Warburg effect, promotes extremely rapid cell proliferation and tumor growth.
http://www.sciencedaily.com/releases/2008/03/080312141243.htm
Fundamental Process Of Tumor Growth Explained
ScienceDaily （Mar. 13, 2008） — Nearly 80 years ago, scientist Otto Warburg observed that cancer cells perform energy metabolism in a way that is different from normal adult cells. Many decades later, this observation was exploited by clinicians to better visualize tumors using PET （positron emission technology） imaging. But it has not been known exactly how tumor cells perform this alternate metabolic feat, nor was it known if this process was essential for tumor growth.
