She told me privately and broke down in tears at the office.
她私下告訴了我這件事，然後在辦公室失聲痛哭。
從那以後，檢測和化療就成了一場噩夢。有時候你能聯繫到人……
有時候你卻聯繫不上。這麼多人被大型製藥公司之類的公司傷害了。
如果我們得不到正義……那麼 5 年後、10 年後或 20 年後我們在哪裡
都無關緊要。那些責任人必須付出代價
It’s hard realizing sometimes you made the wrong decision…
About two years ago, I have a co-worker, whom I care about tell me her and her husband were going to get the boosters one work afternoon. Her husband didn’t want to get it, but she kind of wears the pants in that… pic.twitter.com/ZGAt1RFH0T
I know many were scared, I know many were tricked,
I know many just went along with it all to get on with
their lives…
At the end of the day these people choose to believe
a blatant lie.
These people choose to trade immune systems for donuts These people choose to allow their government to censor, to cancel, and to jail anyone saying different. To me that makes these people dangerous because what will they do next time? If they are scared enough? If they are squeezed enough by leadership?
致癌機制 SV40 在動物中的致癌能力源於其 T 抗原：大 T 抗原（標籤）：結合併滅活 p53 和 pRb，破壞細胞周期調控和細胞凋亡。它還激活信號通路（例如 ERK、AP-1）並通過與 BUB-1 相互作用誘導 DNA 損傷。小 T 抗原（標籤）：通過抑制蛋白磷酸酶 2A、上調 Notch1 和誘導某些細胞中的端粒酶來增強標籤的轉化活性。雖然它對腫瘤誘導並非必需，但它有助於完全轉化。
病毒持久性：在倉鼠中，SV40 不會複製，而是以游離狀態持續存在或整合到宿主基因組中，從而導致持續的致癌基因表達。在小鼠中，病毒 DNA 可以在細胞質中持續存在並進行一定程度的整合，從而導致易感菌株的腫瘤形成。肇事逃逸機制：
一些研究表明，SV40 可以引發腫瘤，然後消失，不留下可檢測的病毒 DNA 或蛋白質，這使其在腫瘤中的檢測變得複雜。與人類癌症的相關性雖然 SV40 能可靠地誘發動物腫瘤，但其在人類癌症中的作用仍存在爭議：支持證據：已在人類腫瘤（例如間皮瘤、腦瘤、骨肉瘤、淋巴瘤）中檢測到 SV40 DNA 和 T 抗原，患病率為 6-60%。激光顯微切割已證實惡性細胞中存在 SV40，但不存在於鄰近的正常組織中。
Dr. Aseem Malhotra calls for urgent suspension of mRNA COVID vaccines: A bold stand for public health
In an interview with Channel 4, renowned cardiologist and public health advocate Dr. Aseem Malhotra delivered a powerful message about the urgent need to reassess the use of… pic.twitter.com/52fTyb9Cl4
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