She told me privately and broke down in tears at the office.
她私下告诉了我这件事，然后在办公室失声痛哭。
从那以后，检测和化疗就成了一场噩梦。有时候你能联系到人……
有时候你却联系不上。这么多人被大型制药公司之类的公司伤害了。
如果我们得不到正义……那么 5 年后、10 年后或 20 年后我们在哪里
都无关紧要。那些责任人必须付出代价
It’s hard realizing sometimes you made the wrong decision…
About two years ago, I have a co-worker, whom I care about tell me her and her husband were going to get the boosters one work afternoon. Her husband didn’t want to get it, but she kind of wears the pants in that… pic.twitter.com/ZGAt1RFH0T
I know many were scared, I know many were tricked,
I know many just went along with it all to get on with
their lives…
At the end of the day these people choose to believe
a blatant lie.
These people choose to trade immune systems for donuts These people choose to allow their government to censor, to cancel, and to jail anyone saying different. To me that makes these people dangerous because what will they do next time? If they are scared enough? If they are squeezed enough by leadership?
致癌机制 SV40 在动物中的致癌能力源于其 T 抗原：大 T 抗原（标签）：结合并灭活 p53 和 pRb，破坏细胞周期调控和细胞凋亡。它还激活信号通路（例如 ERK、AP-1）并通过与 BUB-1 相互作用诱导 DNA 损伤。小 T 抗原（标签）：通过抑制蛋白磷酸酶 2A、上调 Notch1 和诱导某些细胞中的端粒酶来增强标签的转化活性。虽然它对肿瘤诱导并非必需，但它有助于完全转化。
病毒持久性：在仓鼠中，SV40 不会复制，而是以游离状态持续存在或整合到宿主基因组中，从而导致持续的致癌基因表达。在小鼠中，病毒 DNA 可以在细胞质中持续存在并进行一定程度的整合，从而导致易感菌株的肿瘤形成。肇事逃逸机制：
一些研究表明，SV40 可以引发肿瘤，然后消失，不留下可检测的病毒 DNA 或蛋白质，这使其在肿瘤中的检测变得复杂。与人类癌症的相关性虽然 SV40 能可靠地诱发动物肿瘤，但其在人类癌症中的作用仍存在争议：支持证据：已在人类肿瘤（例如间皮瘤、脑瘤、骨肉瘤、淋巴瘤）中检测到 SV40 DNA 和 T 抗原，患病率为 6-60%。激光显微切割已证实恶性细胞中存在 SV40，但不存在于邻近的正常组织中。
Dr. Aseem Malhotra calls for urgent suspension of mRNA COVID vaccines: A bold stand for public health
In an interview with Channel 4, renowned cardiologist and public health advocate Dr. Aseem Malhotra delivered a powerful message about the urgent need to reassess the use of… pic.twitter.com/52fTyb9Cl4
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